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ToolsCompareCardiogen vs Follistatin 315

Cardiogen vs Follistatin 315

Side-by-side comparison of key properties, dosing, and research.

Anti-Aging & Longevity
Cardiogen
Anabolic & IGF
Follistatin 315
Summary
Cardiogen is a tetrapeptide bioregulator (Ala-Glu-Asp-Arg) developed by Professor Vladimir Khavinson. It is a tissue-specific bioregulator for the heart and myocardium, designed to normalize cardiomyocyte function and support cardiac tissue regeneration. Research has demonstrated cardioprotective effects, improved cardiac rhythm, and benefits in recovery from ischemic injury.
Follistatin 315 is a splice variant isoform of follistatin produced by alternative mRNA processing. Unlike Follistatin 344 which is tethered to cell surfaces via heparan sulfate proteoglycans, FST-315 circulates freely in the bloodstream and has broader systemic distribution. It is the predominant circulating form and exerts systemic myostatin inhibition as well as FSH suppression, making it relevant to both muscle growth and reproductive endocrinology.
Half-Life
Short (minutes); gene-regulatory effects persist longer
~3–5 hours (longer systemic circulation vs FST-344)
Admin Route
SubQ, Oral
SubQ, IM
Research
Typical Dose
10 mg per day
No established human dosing protocol
Frequency
Daily for 10–30 days
Research use only
Key Benefits
  • Cardioprotective effects on myocardial tissue
  • Normalization of cardiomyocyte protein synthesis
  • May improve cardiac rhythm and conduction
  • Support for recovery from ischemic cardiac events
  • Anti-aging effects on heart tissue
  • Potential reduction in cardiac fibrosis
  • Often combined with Epithalon for comprehensive cardiovascular longevity support
  • Systemic myostatin inhibition for whole-body muscle growth
  • Freely circulating — broader tissue distribution than FST-344
  • Strong FSH-suppressive activity useful in certain hormonal protocols
  • Potential for greater anabolic effect across multiple muscle groups simultaneously
  • May be more relevant to reproductive endocrinology applications
  • Studied in gene therapy approaches for muscular dystrophy
Side Effects
  • Generally well tolerated in available research
  • Mild injection site reactions
  • No significant adverse cardiovascular events reported at standard doses
  • Systemic FSH suppression — significant concern for fertility
  • Greater potential for off-target effects vs FST-344 due to systemic distribution
  • Limited human safety data
  • Potential cardiac hypertrophy with prolonged high-dose exposure
Stacks With