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ToolsCompareAdamax vs Follistatin 315

Adamax vs Follistatin 315

Side-by-side comparison of key properties, dosing, and research.

Cognitive Enhancement
Adamax
Anabolic & IGF
Follistatin 315
Summary
Adamax is a synthetic neuropeptide related to brain-derived neurotrophic factor (BDNF) signaling pathways. It is explored for cognitive enhancement, neuroprotection, and mood support, with proposed mechanisms involving TrkB receptor activation and enhancement of neuroplasticity similar to endogenous BDNF.
Follistatin 315 is a splice variant isoform of follistatin produced by alternative mRNA processing. Unlike Follistatin 344 which is tethered to cell surfaces via heparan sulfate proteoglycans, FST-315 circulates freely in the bloodstream and has broader systemic distribution. It is the predominant circulating form and exerts systemic myostatin inhibition as well as FSH suppression, making it relevant to both muscle growth and reproductive endocrinology.
Half-Life
Estimated 1-3 hours (short; peptide degradation)
~3–5 hours (longer systemic circulation vs FST-344)
Admin Route
Subcutaneous, Intranasal (research)
SubQ, IM
Research
Typical Dose
200-400 mcg per dose
No established human dosing protocol
Frequency
Once daily or every other day
Research use only
Key Benefits
  • Proposed enhancement of learning and memory consolidation
  • Neuroprotective via BDNF-TrkB pathway support
  • May improve mood and resilience to stress
  • Potential support for neurogenesis
  • Cognitive clarity and focus enhancement (reported anecdotally)
  • Explored for neurodegeneration and age-related cognitive decline
  • Systemic myostatin inhibition for whole-body muscle growth
  • Freely circulating — broader tissue distribution than FST-344
  • Strong FSH-suppressive activity useful in certain hormonal protocols
  • Potential for greater anabolic effect across multiple muscle groups simultaneously
  • May be more relevant to reproductive endocrinology applications
  • Studied in gene therapy approaches for muscular dystrophy
Side Effects
  • Limited human safety data; largely anecdotal reports
  • Possible headache or mild overstimulation
  • Sleep disruption with late-day dosing
  • Unknown long-term safety profile
  • Systemic FSH suppression — significant concern for fertility
  • Greater potential for off-target effects vs FST-344 due to systemic distribution
  • Limited human safety data
  • Potential cardiac hypertrophy with prolonged high-dose exposure
Stacks With